[1] DUAN H B, HU J K, TAN Q J, et al. Systematic characterization of generation and management of e-waste in China[J]. Environmental Science and Pollution Research, 2016, 23(2):1929-1943.
[2] ZENG X L, GONG R Y, CHEN W Q, et al. Uncovering the recycling potential of "New" WEEE in China[J]. Environmental Science and Technology, 2016,50(3):1347-1358.
[3] QIU Y W, LIN D, LIU J Q, et al. Bioaccumulation of trace metals in farmed fish from South China and potential risk assessment[J]. Ecotoxicology and Environmental Safety, 2011, 74(3):284-293.
[4] ZHANG Q, YE J J, CHEN J Y, et al. Risk assessment of polychlorinated biphenyls and heavy metals in soils of an abandoned e-waste site in China[J]. Environmental Pollution, 2014, 185:258-265.
[5] WU Q H, LEUNG J Y, GENG X H, et al. Heavy metal contamination of soil and water in the vicinity of an abandoned e-waste recycling site:Implications for dissemination of heavy metals[J]. Science of the Total Environment, 2015, 506:217-225.
[6] HUO X, PENG L, XU X J, et al. Elevated blood lead levels of children in Guiyu, an electronic waste recycling town in China[J]. Environmental Health Perspectives, 2007, 115(7):1113-1117.
[7] ZHENG L K, WU K S, LI Y, et al. Blood lead and cadmium levels and relevant factors among children from an e-waste recycling town in China[J]. Environmental Research, 2008, 108:15-20.
[8] 赵静, 朱祥伟, 徐挺, 等. BDE17及OH-BDE17暴露对斑马鱼幼鱼的运动行为效应[J]. 环境化学, 2015, 34(7):1239-1245. ZHAO J, ZHU X W, XU T, et al. Locomotor behavior effect in zebrafish larvae after exposure to BDE17 and OH-BDE17[J]. Environmental Chemistry, 2015, 34(7):1239-1245(in Chinese).
[9] VALERIA D G S, ADILSON P S, JHONNES M S T, et al. Metabolic and behavior changes in surubim acutely exposed to a glyphosate-based herbicide[J]. Archives of Environmental Contamination and Toxicology, 2014, 67(4):659-667.
[10] IRONS T D, MACPHAIL R C, HUNTER D L, et al. Acute neuroactive drug exposures alter locomotor activity in larval zebrafish[J]. Neurotoxicology and Teratology, 2010, 32(1):84-90.
[11] ABDULMAJEED W I, SULIEMAN H B, ZUBAYR M O, et al. Honey prevents neurobehavioural deficit and oxidative stress induced by lead acetate exposure in male wistar rats:A preliminary study[J]. Metabolic Brain Disease, 2016, 31(1):37-44.
[12] AKTAR S, JAHAN M, ALAM S, et al. Individual and combined effects of arsenic and lead on behavioral and biochemical changes in mice[J]. Biological Trace Element Research, 2017, 177(2):288-296.
[13] MANSOURI M T, CAULI O. Motor alterations induced by chronic lead exposure[J]. Environmental Toxicology and Pharmacology, 2009, 27:307-313.
[14] BELLINGER D C. Very low lead exposures and children's neurodevelopment[J]. Current Opinion in Pediatrics, 2008, 20(2):172-177.
[15] RICE C, GHORAI J K, ZALEWSKI K, et al. Developmental lead exposure causes startle response deficits in zebrafish[J]. Aquatic Toxicology, 2011, 105(3-4):600-608.
[16] CHEN J, CHEN Y, LIU W, et al. Developmental lead acetate exposure induces embryonic toxicity and memory deficit in adult zebrafish[J]. Neurotoxicology and Teratology, 2012, 34(6):581-586.
[17] ZHAO J, XU T, YIN D Q. Locomotor activity changes on zebrafish larvae with different 2,20,4,40-tetrabromodiphenyl ether (PBDE-47) embryonic exposure modes[J]. Chemosphere, 2014, 94:53-61.
[18] WESTERFIELD M. A guide for the laboratory use of zebrafish (Danio rerio)[M]. Eugene, OR:University of Oregon Press, 2000.
[19] 潘睿捷, 黄文平, 张斌, 等. 斑马鱼幼鱼运动行为测试评价饮用水安全[J]. 生态毒理学报, 2016, 11(4):18-25. PAN R J, HUANG W P, ZHANG B, et al. Toxicity assessment of drinking water using zebrafish swimming behavior tests[J]. Asian Journal of Ecotoxicology, 2016, 11(4):18-25(in Chinese).
[20] ZHANG B, CHEN X L, PAN R J, et al. Effects of three different embryonic exposure modes of 2,2',4,4'-tetrabromodiphenyl ether on the path angle and social activity of zebra fish larvae[J]. Chemosphere, 2017, 169:542-549.
[21] ZHU B R, WANG Q W, WANG X F, et al. Impact of co-exposure with lead and decabromodiphenyl ether(BDE-209) on thyroid function in zebrafish larvae[J]. Aquatic Toxicology, 2014, 157:186-195.
[22] 张小晶. 胚胎期铅暴露对斑马鱼NMDA受体表达及行为的影响[D]. 温州:温州医学院, 2012. ZHANG X J. Effects on expression of NMDA receptor and behavior in zebrafish exposed to lead during embryonic period[D]. Wenzhou:Wenzhou Medical College, 2012(in Chinese).
[23] ZHU B R, WANG Q W, SHI X J, et al. Effect of combined exposure to lead and decabromodiphenyl ether on neurodevelopment of zebrafish larvae[J]. Chemosphere, 2016, 144:1646-1654.
[24] WIRBISKY S E, WEBER G J, LEE J W, et al. Novel dose-dependent alterations in excitatory GABA during embryonic development associated with lead (Pb) neurotoxicity[J]. Toxicology Letters, 2014, 229(1):1-8.
[25] TU H W, FAN C J, CHEN X H, et al. Effects on cadmium, manganese, and lead on locomotor activity and neurexin 2A expression in zebrafish[J]. Environmental Toxicology and Chemistry, 2017, 38(8):2147-2154.
[26] CHEN X J, HUANG C J, WANG X C, et al. BDE-47 disrupts axonal growth and motor behavior in developing zebrafish[J]. Aquatic Toxicology, 2012, 120-121:35-44.
[27] CHEN L G, HUANG C J, HU C Y, et al. Acute exposure to DE-71:Effects on locomotor behavior and developmental neurotoxicity in zebrafish larvae[J]. Environmental Toxicology and Chemistry, 2012, 31(10):2338-2344.
[28] CHEN L G, HUANG Y B, HUANG C J, et al. Acute exposure to DE-71 causes alterations in visual behavior in zebrafish larvae[J]. Environmental Toxicology and Chemistry, 2013, 32(6):1370-1375.
[29] XU T, ZHAO J, Yin D Q, et al. High-throughput RNA sequencing reveals the effects of 2,2',4,4' -tetrabromodiphenyl ether on retina and bone development of zebrafish larvae[J]. BMC Genomics, 2015, 16(1):1-12.
[30] ZHAO J, XU T, YIN D Q, et al. The regulatory roles of microRNA in effects of 2,2' 4,4'-tetrabromodiphenyl ether (BDE47) on the transcriptome of zebrafish larvae[J]. Plos One, 2017,12(1):e0169599.
[31] XU T, LIU Y, PAN R J, et al. Vision, color vision, and visually guided behavior:The novel toxicological targets of 2,2',4,4'-tetrabromodiphenyl ether (BDE-47)[J]. Environmental Science & Technology Letters, 2017, 4(4):132-136.
[32] VAN LEEUWEN J L, VOESENEK C J, MULLER U K. How body torque and Strouhal number change with swimming speed and developmental stage in larval zebrafish[J]. Journal of the Royal Society Interface, 2015, 12(110):1-11.
[33] MULLER U K, VAN LEEUWEN J L. Swimming of larval zebrafish ontogeny of body waves and implications for locomotory development[J]. Journal of Experimental Biology, 2004, 207(5):853-868.
[34] BUDICK S A, OMALLEY D M. Locomotor repertoire of the larval zebrafish swimming, turning and prey capture[J]. Journal of Experimental Biology, 2000, 203(17):2565-2579.
[35] CRETON R. Automated analysis of behavior in zebrafish larvae[J]. Behavioural Brain Research, 2009, 203:127-136.
[36] COLWILL R M, CRETON R. Imaging escape and avoidance behavior in zebrafish larvae[J]. Reviews in the Neurosciences, 2011, 22:63-73.
[37] 邹苏琪, 殷梧, 杨昱鹏. 斑马鱼行为学实验在神经科学中的应用[J].生物化学与生物物理进展, 2009, 36(1):5-12. ZOU S Q, YIN W, YANG Y P, et al. The ethology application of zebrafish in neuroscience[J]. Progress in Biochemistry and Biophysics, 2009, 36(1):5-12(in Chinese).
[38] BAILEY J M, OLIVERI A N, KARBHARI N, et al. Persistent behavioral effects following early life exposure to retinoic acid or valproic acid in zebrafish[J]. Neurotoxicology, 2016, 52:23-33.